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Semax is a synthetic heptapeptide (Met-Glu-His-Phe-Pro-Gly-Pro), an ACTH(4-7) fragment extended with Pro-Gly-Pro, which slows enzymatic breakdown. Unlike full ACTH it does not drive corticosteroid release, and published literature ties it to BDNF and NGF expression and to monoaminergic and melanocortin receptor systems. It is a research peptide, not an approved medicine, so it suits laboratory study rather than self-treatment. Rival nootropic peptides like Selank are often discussed alongside it, but Semax's ACTH lineage and degradation-resistant tail are what set it apart.
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Semax is a synthetic heptapeptide with the sequence Met-Glu-His-Phe-Pro-Gly-Pro. It consists of the ACTH(4-7) fragment extended at the C-terminus by the tripeptide Pro-Gly-Pro.
The Pro-Gly-Pro addition makes it resistant to enzymatic degradation, compared with the unmodified ACTH fragment. Unlike full-length ACTH, the 4-7 fragment does not stimulate corticosteroid release.
In published literature, Semax has been characterized in connection with brain-derived neurotrophic factor and nerve growth factor expression, and with monoaminergic and melanocortin receptor systems.
